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| (especial para SIIC © Derechos reservados) |
| Gisela Gutiérrez Iglesias* Autora invitada por SIIC |
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![]() Profundizar Referencias bibliográficas |
| Bauleiu EE. Dehydroepiandrosterone (DHEA): a fountain of youth? J Clin Endocrinol Metabol 1:3147-3151, 1996. Chen H, Li D, Saldeen T, Mehta JL. Transforming growth factor-?1 modulates oxidatively modified LDL-induced expression of adheson molecules: role of LOX-1. Circ Res 89:1155-1160, 2001. Ebeling P, Koivisto VA. Physiological importance of dehydroepiandrosterone. Lancet 343:1479-1481, 1994. Ganong FW. Fisiología Médica. 15ª Edición. Editorial El manual moderno. México, D.F. pp. 402-404, 1996. Holvoet P, Collen D. Beta-VLDL hypercholesterolemia relative to LDL hypercholesterolemia is associated with higher levels of oxidized lipoproteins and a more rapid progression of coronary atherosclerosis in rabbits. Arterioscler Thromb Vasc Biol 17:2376-2382, 1997. Khorram O, Vu L, Yen SS. Activation of immune function by dehydroepiandrosterone (DHEA) in age-advanced men. J Gerontol A Biol Sci Med Sci 52:M1-7, 1997. Li D, Mehta JL. Antisense to LOX-1 inhibits oxidized LDL-mediated upregulation of monocyte chemoattractant protein-1 and monocyte adhesion to human coronary artery endothelial cells. Circulation 102:2889-2895, 2000. Nestler JE, Barlascini CO, Clore JN, Blackard WG. Dehydroepiandrosterone reduces serum low density lipoprotein levels and body fat but does not alter insulin sensitivity in normal men. J Clin Endocrinol Metab 66:57-61, 1998. Nilsson J Atherosclerosis: the molecular background. Lakartidningen 88:127-129, 1991. Regelson W, Kalimi M, Loria R. DHEA: Some thoughts as to its biologic and clinical action. In: Kalimi M, Regelson W. (Eds.) The biologic role of dehydroepiandrosterone (DHEA). Walter de Gruyter & Co, Berlin, New York, pp. 405-445, 1990. Sanders M. Molecular and cellular concepts in atherosclerosis. Pharmacol Ther 61:109-153, 1994. Schwartz AG, Pashko L, Whitcomb JM. Inhibition of tumor development by dehydroepiadrosterone and related steroids. Toxicol Pathol 14:357-362, 1986. Schwartz C, Valente A, Sprague E, Kelley J, Nerem R. The pathogenesis of atherosclerosis: an overview. Clin Cardiol, 14:11-16, 1991. Tchernof A, Labrie F, Belanger A, Despres JP. Obesity and metabolic complications: contribution of dehydroepiandosterone and other steroid hormones. J Endocrinol 150(suppl):155-164, 1996. Watson RR, Huls A, Araghinuam M, Chung S. Dehydroepiandrosterone and diseases of aging. Drugs Aging 9:274-291, 1996. |
| Otros artículos de Gisela Gutiérrez Iglesias |
| López Marure R, Gutiérrez G, Mendoza C, Ventura JL, Sánchez L, Reyes Maldonado E, Zentella A, Montaño LF. Ceramide promotes death of human cervical tumor cells in the absence of biochemical and morphological markers of apoptosis. Biochemical Biophysical Research Communications 293(3):1028-1036, 2002. Páez A, Archundia A, Méndez R, Rodríguez E, López Massó F, Aceves JL., Flores L, Montaño LF. A subpopulation of large granular von Willebrand Ag negative and CD105 positive endotelial cells, isolated from abdominal aortic aneurysms, overexpresses ICAM-1 and Fas antigen. Archivos de Cardiología de México 72(2):99-104, 2002. Rodríguez E, López R, Paez A, Massó F, Montaño LF. 17?-estradiol inhibits the adhesion of leukocytes in TNF-alpha stimulated human endothelial cells by blocking IL-8 and MCP-1 secretion, but not its transcription. Life Sciences 71(18):2181-2193, 2002. Zapata E, Ventura JL, De la Cruz K, Rodríguez E, Massó PDF, Montaño LF, López Marure R. Dehidroepiandrosterone inhibits the proliferation of HUVEC by enhancing the expression of p21 and p53, restricting the phosphorylation of RB, and is androgen- and estrogen-receptor independent. FEBS Journal 272:1343-1353, 2005. Sánchez L, Hernández V, López Marure R. Papel de las cadherinas en la metástasis. REB 24(3,4):97-103, 2005. Gayosso V, Montaño LF, López Marure R. DHEA-induced antiproliferative effect in MCF-7 cells is androgen- and estrogen-receptor independent. The Cancer Journal 12(2):160-165, 2006. Alfaro Moreno E, López Marure R, Montiel A, Symond P, Osornio Vargas AR, Rosas I, Clifford J. E-selectin expression in human endothelial cells exposed to PM10: the role of endotoxin and insolucle fraction. Environmental Research. In press. Gutiérrez G, Mendoza C, Montaño LF, López Marure R. Ceramide induces early and late apoptosis in HPV+ cervical cancer cells by inhibiting ROS decay, diminishing the intracellular concentration of glutathione, and increasing NF-kB translocation. Anti-Cancer Drugs. In press. Montiel Dávalos A, Alfaro Moreno E, López Marure R. PM2.5 and PM10 from Mexico city induce the expression of adhesion molecules and the adhesion of monocytic cells to human umbilical vein endothelial cells. Inhalation Toxicology. In press. |
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Acerca del trabajo completo |
| EFECTO ANTIINFLAMATORIO DE LA DESHIDROEPIANDROSTERONA EN CELULAS ENDOTELIALES |
| Título original en castellano LA DESHIDROEPIANDROSTERONA INHIBE LA RESPUESTA INFLAMATORIA INDUCIDA POR EL TNF-ALFA EN CELULAS ENDOTELIALES HUMANAS DE CORDÓN UMBILICAL |
| Autores Gisela Gutiérrez Iglesias1, Criselda Mendoza2, Estrella Zapata3, Angélica Montiel4, Elba Reyes5, Luis Felipe Montaño6 |
| 1 PhD, Departamento de Biología Celular, Instituto Nacional de Cardiología "Ignacio Chávez", Investigadora 2, Centro de Investigación en Enfermedades Infecciosas, Investigadora 3 Maestra en Ciencias, Departamento de Biología Celular, Instituto Nacional de Cardiología "Ignacio Chávez", Investigadora 4 Bióloga, Departamento de Biología Celular, Instituto Nacional de Cardiología "Ignacio Chávez", Estudiante de Maestría 5 PhD, Departamento de Morfología, Escuela Nacional de Ciencias Biológicas, IPN, México, Investigadora 6 PhD, Laboratorio de Inmunología, Departamento de Bioquímica, Facultad de Medicina, UN, Investigador |
| Acceso a la fuente original Atherosclerosis |
| http://www.elsevier.com/wps/find/journaldescription.cws_home/522790/description |
Acceso al texto original completo (full text) |
Acceso al resumen/abstract original |
| El artículo se relaciona estrictamente con las especialidades de siicsalud | |
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